Impact of Diabetes on the Nutritional Status of CKD Patients
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Abstract
Background: Prevalence of malnutrition in Chronic Kidney Disease (CKD) patients is due to their lower appetite level. Diabetic patients experience polyphagia. Hence it is necessary to understand the impact of diabetes on the nutritional status of CKD patient.
Aim: To compare the nutritious status between the CKD patients with and without diabetes.
Objectives: To find out the impact of diabetes on the nutritional status of CKD patients.
Method and Materials: A prospective random sampling method was adopted to select the subjects. Eighty CKD patients were divided equally into two groups. Tool which has been used to collect the data was SOAP format.
Results: While comparing BMI between group 1(CKD without diabetes) and group 2(CKD with diabetes) it was noticed that the percentage of normal nourished subjects in group 2 were greater than that of group1. About 63% and 25% of subjects with CKD in group1 had normal BMI and grade 1 undernourished respectively. In group 2, about 75% and 10% of patients had normal BMI and grade 1 over nourished respectively. About 80% of subjects in group 2 were on insulin treatment. About 88% of subjects in group1 were anorexic and about 55% and 25% of subjects in group 2 had polyphagia and normal appetite respectively. The difference in the energy and protein intake between the two groups was statistically significant at p<0.01 level.
Conclusions: It can be concluded from the above study that a better nutritional status was found in the CKD patients with Diabetes Mellitus (DM) than the CKD patients without DM which may be due to their normal appetite/ polyphagia, hence allowing a better food intake among group2 subjects. Weight gain in CKD with DM patients may be due to increase in fat mass which is subsequent to lipogenic effect of insulin.
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Copyright (c) 2017 Mathew AB, et al.

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Coles GA. Body composition in chronic renal failure. Q J Med. 1972; 41: 25-47. Ref.: https://goo.gl/4t1sQ70
Cano NJ, Roth H, Aparicio M, Azar R, Canaud B, et al. Malnutrition in hemodialysis diabetic patients: evaluation and prognostic influence. Kidney Int. 2002; 62: 593-601. Ref.: https://goo.gl/1hWmfX
Warram JH, Gearin G, Laffel L, Krolewski AS. Effect of duration of type I diabetes on the prevalence of stages of diabetic nephropathy defined by urinary albumin/creatinine ratio. J Am Soc Nephrol. 1996; 7: 930-937. Ref.: https://goo.gl/QGXeLJ
Gross JL, de Azevedo MJ, Silveiro SP, Canani LH, Caramori ML, et al. Diabetic nephropathy: diagnosis, prevention, and treatment. Diabetes Care. 2005; 28: 164-176. Ref.: https://goo.gl/5HRcSa
Hall JE, Guyton AC. Guyton and Hall textbook of medical physiology, Insulin, glucagon, and diabetes mellitus, 12th edition. Saunders Elsevier. 2011. Ref.: https://goo.gl/h8zCDW
Mudaliar S, Edelman SV. Insulin Therapy in Type 2 Diabetes. Endocrinology and Metabolism Clinics. 2001; 30: 1-32. Ref.: https://goo.gl/J39P8w
Jacob AN, Salinas K, Adams-Huet B, Raskin P. Potential causes of weight gain in type 1 diabetes mellitus. Diabetes Obes Metab. 2006; 8: 404-411. Ref.: https://goo.gl/QkI1Ey
http://www.diabetes-kidney.org/diabetic-nephropathy/631.html
Alebiosu CO, Ayodele OE. The global burden of chronic kidney disease and the way forward. Ethn Dis. 2005; 15: 418-423. Ref.: https://goo.gl/5TC0T0
Garrow JS. Energy balance and obesity in man, (2nd edn). Elsevier Holland. 1978. Ref.: https://goo.gl/mcMGNR
Jacob AN, Salinas K, Adams-Huet B, Raskin P. Weight gain in type 2 diabetes mellitus. Diabetes Obes Metab. 2007; 9: 386-393. Ref.: https://goo.gl/K9veHf